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Patano and colleagues suggest that this decrease in energy availability might compromise osteoblast activity and bone remodeling in an age-related manner [152]
Jieduquyuziyin prescription enhances CD11a and CD70 DNA methylation of CD4(+) T cells via miR-29b-sp1/DNMT1 pathway in MRL/lpr mice
Individual differences in susceptibility to idiosyncratic drug-induced liver injury may reflect genetic differences in either (a) alternate metabolic pathways by which a drug is converted to different (potentially hepatotoxic) metabolites, (b) the ability of the individual to detoxify the toxic intermediates, (c) an underlying immunologic or allergic reaction, or (d) an individual's tolerance or ability to adapt to hepatocellular injury (mechanisms unknown) with resolution of injury despite continued medication administration.20, 135 Oral medications with substantial hepatic metabolism are more likely to be associated with adverse hepatic events in humans, presumably because of hepatic generation of reactive toxic metabolites
10.1034/j.1600-079x.2000.280407.x 77 MartnM.MacasM.LenJ.EscamesG.KhaldyH.Acua-CastroviejoD
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