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S-Nitrosylation of 1-antitrypsin triggers macrophages toward inflammatory phenotype and enhances intra-cellular bacteria elimination

reported that a panel of non-small cell lung cancer (NSCLC) cell lines exhibited heterogeneous responses to cystine starvation despite the consistent intracellular cysteine deletion and impaired GSH biosynthesis 12

However, further research is planned to improve our understanding of their effects on obesity-related comorbidities and the underlying mechanism, whether involving direct effects on target tissues or mediated by improvement of adiposity, glucose levels and other CV risk factors