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In the context of UAs protective mechanisms, BDNF and NGF are believed to contribute to reduced brain damage and improved functional outcome in a model of focal ischemic stroke [145, 172]

The CD4+ T lymphocytes are the main targets of HTLV-1 infection, but it has been shown that other leukocytes and glial cells are also infected [19, 20, 21]

This, in turn, supports DNA repair processes and reduces neuroinflammation, potentially improving cognitive function and slowing the progression of neurodegenerative conditions