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Moreover, the hyperhomocysteinemia associated with an increased cardiovascular risk is stronger in non-insulin-dependent diabetes mellitus than in normal subjects (Hoogeveen et al, 1998)

[ 81 , 82 ] Under oxidative stress conditions, BACH1 is regulated by ROS, which inactivates BACH1 through oxidizing its cysteine residue, [ 83 ] leading to the upregulation of antioxidant genes like HO1, NQO1, GCLC, GCLM, and SLC7A11

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