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Cysteine plays a role in the sulfation cycle, acting as a sulfur donor in phase II detoxification and as a methyl donor in the conversion of homocysteine to methionine [13-15]
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This might even explain why the changes in HbA1c observed in DG have shown limited effect sizes
RT-PCR analysis revealed that the administration of silymarin upregulates genes involved in GSH metabolism, as well as in glutamate and citrate metabolisms, which indirectly could increase GSH [293]
