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361 In the context of ataxia-telangiectasia mutated (ATM) deficiency, treatment with the permeable thiol antioxidant N-acetyl-l-cysteine (NAC) was shown to restore the reconstitution capacity of HSCs, thereby preventing bone marrow failure

However, it should be noted that some studies have failed to detect significant generation of new neurons post-developmentally in humans (Cipriani et al., 2018

The second step in ATXN3 aggregation, exclusively dependent on the polyQ expansion, leads to the generation of mature and SDS-resistant ATXN3 fibrils [38,39,40,41]

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