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As mentioned, infectious mononucleosis caused by EBV is a risk factor for various autoimmune diseases (487, 497), and may play a potential role in ME/CFS (140, 498500)

These models allow the investigation of molecular pathways underlying tumorigenesis and cellular processes driving nodule formation, providing critical insights into the pathophysiology of thyroid cancer

Moreover, phospho-BCL-XL (Ser62) stabilizes G2 arrest by binding to and co-localizing with Cdk1 (cdc2) in nucleolar structures to avoid rapid nucleolar disassembly associated with mitosis onset, playing an unexpected role in the G2 phase checkpoint downstream of DNA damage distinct from its anti-apoptotic function 22

Prior to 2019, no studies adequately addressed these limitations