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In mice, the acute systemic inhibition of GSH synthesis by L-buthionine-S-R-sulfoximine (BSO) triggers a protective response and a subsequent increase in the CNS GSH content

It is quintessential for optimum cellular functioning and detoxification

Collectively, the animal studies of moderate brain injuries suggest that such injuries cause a release of signaling molecules which results in a disruption of the ionic balance across the cell membrane immediately after the injury event