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Current research indicates that the cell cycle arrest depends on the activity of cyclin-dependent kinase inhibitors (CDK) p16 and p21, often regulated by the main tumor suppressor protein p53, whose elevated level is frequently used as a marker of senescence detection [21]

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Another study published in Behavioural Brain Research reported that Dihexa administration reversed memory loss in a mouse model of Alzheimers disease, suggesting its potential in neurodegenerative disease conditions

(2) Loren Pickart and Anna Margolina (2018) Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data International Journal of Molecular Sciences, 2018 Jul 7, Volume 19 (Issue 7), Page 1987