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These mechanisms are not fully understood but include central nervous system carbohydrate reduction and glycolysis inhibition, changes in neuronal excitability mediated by ATP-sensitive potassium channels through alterations in mitochondrial function, inhibition of the mammalian target of rapamycin (mTOR) pathway, and inhibition of glutamatergic excitatory synaptic transmission [72]

In view of the role of Se deficiency in promoting thyroid tumorigenesis and progression, appropriate Se supplementation for differentiated thyroid cancer has been proposed to delay the disease progression and improve the prognosis (98).Kato, M