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SHP type-1 (SHP-1) is involved in inducing apoptosis by increasing pro-apoptotic proteins such as the p53-Bax-caspase-3 pathway and by increasing JNK expression resulting in inhibition of anti-apoptotic proteins (Fig

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One of the mechanisms by which senescent cells escape from the immune response is made through the overexpression of the histocompatibility antigen alpha chain E (HLA-E) that bind to NKG2A, an inhibitor receptor expressed on Natural Killer cells (NK) 54 and CD8 + T cells, leading to the inhibition of SnCs recognition