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The CNS GLP- 1 receptors are not necessary for the physiological regulation of blood glucose levels by endogenous GLP- 1 or the pharmacological control of glucose homeostasis that depends on GLP- 1 receptors, although further research is needed to investigate the possible role of GLP- 1 receptors expressed outside the beta cell, including within the enteric and peripheral nervous systems, in improving beta cell function through enteral glucose mediation [70, 84]

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GHK-Cu has also been linked to several other processes that support tissue creation and has been found to: Stimulate the synthesis of collagen in skin cells [3] Increase the accumulation of protein [3] Increase synthesis of decorin [4] Stimulate the synthesis of metalloproteinases Suppress the production of the inflammatory cytokine interleukin-6 [2] Stimulate antioxidant gene activity [2] Essentially, GHK-Cu is believed to help the body break down old tissue, repair DNA, and create new, healthier tissue