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There is a correlation between TLR4 activation and CP-induced neurotoxicity, as well as the following surplus output of pro-inflammatory cytokines and their important role in the pathophysiology of the cognitive impairment that is associated with this condition, according to a study that was conducted not too long ago (Ren et al

Cell Microbiol (2015) 17:13145.10.1111/cmi.12343 49 ImaiYKubaKNeelyGGYaghubian-MalhamiRPerkmannTVan LooGet alIdentification of oxidative stress and toll-like receptor 4 signaling as a key pathway of acute lung injury

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Copper signaling is believed to play a role in GHK-Cus biological activity, and a specific level of copper is required for stem cells to function correctly [5]