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Previous studies indicated that lysosomal iron is released and translocated into the mitochondria contributing to MPT in APAP-induced injury ( 2+ uniporter MCFU plays a key role in APAP-induced oxidative stress, mitochondrial dysfunction, and hepatocellular death ( 2+ from lysosomes followed by uptake into mitochondria via MCFU is a key mechanism in APAP-mediated hepatotoxicity

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Beyond redox regulation, GSH is involved in several essential cellular processes, including the synthesis of DNA and proteins, modulation of enzyme function, gene expression regulation, signal transduction, and both intracellular and transmembrane transport [13]

To this end, 32D cells, a mouse myeloid progenitor line which proliferation is dependent on interleukin-3 (IL-3) 26 , were employed for WT- and T315I-Bcr-Abl transformation