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In the Turkish population study [22], the authors suggested that the GSTM1 gene may be a useful marker in the prediction of T2DM susceptibility

The outcome suggested that a transient and limited increase in gut permeability compared with the unregulated breach of the gut barrier function due to the TJ deregulation and epithelial cell apoptosis can rather lead to a dominant regulatory immune response to gut microbiota antigens and to a net anti-inflammatory effect

Oral bioavailability would then be higher than predicted, based on consideration of hepatic extraction ratio and f H

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