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Proper NO signaling in the body is imperative for maintaining homeostasis (healthy balance)

Interestingly, another mechanism was proposed as an alternative way to stabilize NRF2 through the interaction between the autophagy adaptor protein p62/sequestosome-1 (p62/SQSTM1) and KEAP1, which leads to the sequestration of KEAP1 in the autophagy-deficient cells and enables NRF2 accumulation [20]

In general, oxidative stress results from the abnormal production of ROS and the loss of cellular antioxidant capacity

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