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Cells undergoing cuproptosis exhibit unique biochemical, genetic, metabolic, and morphological features that distinguish them from other recognized forms of cell death, such as apoptosis, pyroptosis, necroptosis, and ferroptosis [13, 14] (Table 1)

Latency times were recorded, with 0 seconds assigned if the animal was unable to grasp the rod [1]

This would lead to the accumulation of senescent cells, the persistence of their pro-inflammatory SASP, and the chronic inflammation and tissue disruption seen in the disease, further reinforcing the pathological cycle

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