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NRF2 enhances the cells capacity to manage oxidative stress and ionizing radiation, as well as other exogenous and endogenous challenges, by regulating the expression of various antioxidant genes, such as glutathione S-transferase, glutathione reductase, and NAD(P)H quinone oxidoreductase [1]

We found that inhibiting EAAT2, which mediates glutamate uptake into astrocytes, a trigger for lactate production and release into the extracellular space 22,40 , only mildly affected the optogenetic stimulation effect on synchronized activity in a concentration dependent manner

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