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Reductions in glutathione synthesis and tissue glutathione concentrations in aged animals (compared to younger ones) are suggestive of a potentially lower ability to respond to oxidative stress or toxin exposure (28)

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Co-culture experiments showed that AMSCs can significantly reduce JNK activation (mitochondrial stress) and the expression of H2AX (DNA damage) caused by APAP toxicity and increase the expression of DDIT4 in AML12 cells and primary hepatocytes (Fig