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Although deferiprone has been authorized for only limited diseases, its ability to interfere with iron lossinduced mitophagy highlights its therapeutic potential in treating SLE and thus deserves further investigation (95)

Multiple reports have shown that G6PD-deficient infants are significantly predisposed to neonatal jaundice (6264) and that it is more likely to be severe enough to cause kernicterus

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[42] These authors have shown that partial depletion of GSH with either phorone or DEM results in a four- to five-fold increase in hepatic gamma glutamyl cysteinyl synthetase RNA levels