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A GST was induced in the resistant cultivar but its expression was hardly detectable in susceptible plants, suggesting that this GST may play a significant role in anthracnose resistance (Li et al., 2013)

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Both population and in vivo evidence indicate that oral Vitamin D supplementation increases cathelicidin levels in specific skin lesions, while topical Vitamin D analogues (e.g., calcipotriol) similarly enhance hCAP18/LL-37 expression in wound models, suggesting that correcting Vitamin D axis hyporesponsiveness may help reverse the age-associated phenotype of AMP deficiency and susceptibility [243, 244]