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Edaravone prevents reactive oxygen species from being formed and/or removes them before they can damage neurons, in effect limiting apopotosis of post synaptic terminals as well as the inactivation of EAAT2 receptors in astrocytes and increasing the rate of glutamate re-uptake (Figure 1)

However, all formulations generated reactive oxygen species

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Limited clinical data exist supporting L-carnitine's use in the management of mitochondrial dysfunction related to aging and apoptosis, AIDS-related dementia complex, drug-induced anemia, bipolar depression, cachexia, cardiovascular disease, dyslipidemia, dysthymic disorder, fatigue, drug-induced hepatotoxicity, intermittent claudication, migraine prophylaxis, muscle cramps, and drug-induced peripheral neuropathy