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However, studies to date have been limited by small sample sizes, varying treatment protocols, and frequent recurrences of SIBO, mainly due to gastrointestinal dysmotility that persists in SSc

With the use of the ASOs to target the SCN1A NAT, one study was effectively able to rescue the sense SCN1A gene expression, ameliorate neuronal excitability and seizures in a mouse model of DSan effect that almost entirely recapitulates the rescued phenotypes observed via CRISPRa treatment [74, 75]

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